This first one is pretty close to a verbatim of my quote ...and it wasn't even very hard to find.
LOL...lets take a look, shall we?
Given the multifactorial nature of an individual’s risk, it can be argued that an individual’s familial risk of disease may, in fact, be a better indicator of the many complex interactions among predisposing genetic and environmental factors than can be captured by an individual’s own risk factors.
What does that say exactly?
Given the multifactorial nature of an individual’s risk...It says "given the
MANY, MANY INTER-RELATED nature of an individuals risk.....
So...we are dealing with a risk that has MANY INTER-RELATED factors...right?
Moving on:
it can be argued that an individual’s familial risk of disease may, in fact,HMMMM...."it can be argued" means it is debatable....."risk of disease
MAY, in fact"......MAY=maybe, might, not a fact.
Again...moving along:
"be a better indicator of the many complex interactions among"Hmmmm....."many complex interactions" sounds like the "multifactorial nature" we addressed earlier.
So wher are we up to now?
Given the MANY INTER-RELATED nature of an individuals risk.....it can be DEBATED that an individual’s familial risk of disease MIGHT be a better indicator of the MANY COMPLEX INTERACTIONS among.......
Among what?
Predisposing genetic and environmental factorsAnd we discussed predisposing earlier....predisposing gaurentees NOTHING.
WOW! Impressive!

Rather than dissect the rest of this crap, I'll hit the high points:
Scientists using powerful new genetics research methods have for the first time identified a snippet of DNA common to many people that dramatically increases the chances of developing heart disease.
Does McCain have this "snippet?"
What about Obama?
What if Obama has it and McCain doesn't?
As a result of the atherosclerotic process, cardiovascular disease is due to the interaction between environmental risk factors, such as diet, physical inactivity, smoking, and an individual's genetic makeup. Hundreds of genes are believed to be involved in the process of atherogenesis and the susceptibility to cardiovascular disease. These include genes that regulate lipid metabolism, inflammatory and immune responses, endothelial function, and coagulation. Other genes involved in obesity, insulin resistance, diabetes, elevated homocysteine levels, and hypertension have been identified, but their mechanisms in the atherosclerotic process are not well understood (Lusis, 2003). The genes involved in lipid metabolism have been extensively studied and identified, specifically the gene coding for the low-density lipoprotein (LDL) receptor.
Again we have a plethora of factors here..and again with the genetics that we have NO IDEA as to which candidate possesses them.
Key to my questions last time, something you ignored....was:
Just what weight do you want to add....more significant than sedentary life style, obesity, and smoking?Go ahead and tell us all how extremely important mommy and daddy's health history is to you.....